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Indications of IL1β, IL18 and VEGF cascade in maintenance of homeostasis following transient pulmonary inflammation due to carbon nanoparticle (CNP) challenge in mice

K Ganguly, S Upadhyay, M Irmler, S Takenaka, K Pukelsheim, J Beckers, E Hamelmann, H Schulz, T Stoeger · Pneumologie · Ausgabe 01/2010

DOI 10.1055/s-0029-1247913

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Autor:innenK Ganguly, S Upadhyay, M Irmler, S Takenaka, K Pukelsheim, J Beckers, E Hamelmann, H Schulz, T Stoeger
ZeitschriftPneumologie
Ausgabe01/2010
Jahrgang / SeitenNicht angegeben

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Aims: Carbonaceous nanoparticles possess an emerging source of human exposure. The pulmonary inflammation caused by deposited nanoparticles is a key event for their adverse health effects. Epidemiological studies suggest that individuals with favourable lung physiology are at lower risk for particulate matter associated respiratory diseases. We selected a mouse strain C3H/HeJ (C3) with robust lung physiology and exp…

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Zitierempfehlung

K Ganguly, S Upadhyay, M Irmler, S Takenaka, K Pukelsheim, J Beckers, E Hamelmann, H Schulz, T Stoeger (2010) Indications of IL1β, IL18 and VEGF cascade in maintenance of homeostasis following transient pulmonary inflammation due to carbon nanoparticle (CNP) challenge in mice. Pneumologie (01). https://doi.org/10.1055/s-0029-1247913

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