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Differential expression of PPARalpha, STAT1 and STAT3 in TAA/EtOH-induced liver fibrosis in cannabinoid recepetor 1 knockout mice

K Quint, M Stoll, T Wissniowski, A Agaimy, F Stickel, P Di Fazio, M Ocker · Zeitschrift für Gastroenterologie · Ausgabe 01/2011

DOI 10.1055/s-0030-1269491

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Autor:innenK Quint, M Stoll, T Wissniowski, A Agaimy, F Stickel, P Di Fazio, M Ocker
ZeitschriftZeitschrift für Gastroenterologie
Ausgabe01/2011
Jahrgang / SeitenNicht angegeben

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Aims: CB1 and CB2 are the main receptor for endocannabinoids. An upregulation has been described during chronic liver diseases and a pro-fibrogenic role was shown for CB1. We here applied the TAA/EtOH model of fibrosis induction to determine the underlying signaling pathways in CB1-related fibrosis development. Methods: CB1 knockout mice and C57/B6 wildtype (WT) littermates were treated for 18 weeks with intraperito…

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K Quint, M Stoll, T Wissniowski, A Agaimy, F Stickel, P Di Fazio, M Ocker (2011) Differential expression of PPARalpha, STAT1 and STAT3 in TAA/EtOH-induced liver fibrosis in cannabinoid recepetor 1 knockout mice. Zeitschrift für Gastroenterologie (01). https://doi.org/10.1055/s-0030-1269491

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