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Selective inhibition of chemokine CCL2/MCP-1 reduces experimental pulmonary hypertension

D Kosanovic, BK Dahal, C Vroom, E Bieniek, H Ardeschir Ghofrani, N Weissmann, W Seeger, F Grimminger, S Klussmann, D Eulberg, RT Schermuly · Pneumologie · Ausgabe 05/2013

DOI 10.1055/s-0033-1345048

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Autor:innenD Kosanovic, BK Dahal, C Vroom, E Bieniek, H Ardeschir Ghofrani, N Weissmann, W Seeger, F Grimminger, S Klussmann, D Eulberg, RT Schermuly
ZeitschriftPneumologie
Ausgabe05/2013
Jahrgang / SeitenNicht angegeben

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Abstract-Vorschau

Background: It has been shown previously that the increased levels of pro-inflammatory infiltrates and accumulation of inflammatory cells are present in clinical and experimental pulmonary hypertension (PH), suggesting the role of inflammation in pulmonary vascular remodelling. Literature implicates the involvement of the chemokine CCL2/monocyte chemoattractant protein (MCP)-1 in PH development. This study aimed to…

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Zitierempfehlung

D Kosanovic, BK Dahal, C Vroom, E Bieniek, H Ardeschir Ghofrani, N Weissmann, W Seeger, F Grimminger, S Klussmann, D Eulberg, RT Schermuly (2013) Selective inhibition of chemokine CCL2/MCP-1 reduces experimental pulmonary hypertension. Pneumologie (05). https://doi.org/10.1055/s-0033-1345048

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