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Differential function of IL-17A in cigarette smoke induced lung damage

M Voss, D Wonnenberg, A Kamyschnikow, C Herr, M Menger, R Bals, C Beisswenger · Pneumologie · Ausgabe 02/2014

DOI 10.1055/s-0034-1367900

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Autor:innenM Voss, D Wonnenberg, A Kamyschnikow, C Herr, M Menger, R Bals, C Beisswenger
ZeitschriftPneumologie
Ausgabe02/2014
Jahrgang / SeitenNicht angegeben

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Abstract-Vorschau

Smoking is the main risk factor for the chronic obstructive pulmonary disease (COPD). A recent study demonstrated by quantitative microcomputed tomography (µCT) that IL-17A-deficient mice exposed to cigarette smoke are protected from increases in lung volume and decreased lung density. This suggests that Th-17/IL-17-dependent immune mechanisms mediate smoke-induced lung damage, such as the development of emphysema.…

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Zitierempfehlung

M Voss, D Wonnenberg, A Kamyschnikow, C Herr, M Menger, R Bals, C Beisswenger (2014) Differential function of IL-17A in cigarette smoke induced lung damage. Pneumologie (02). https://doi.org/10.1055/s-0034-1367900

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