CareLit · DOI-Nachweis · Pneumologie
Alternative chloride channels in cystic fibrosis (CF): Functional characterization of SLC26A9 in stably-overexpressing FRT epithelial cells
DOI 10.1055/s-0035-1556599
Bibliografische Angaben
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Autor:innenJJ Salomon, S Spahn, X Wang, J Füllekrug, CA Bertrand, MA Mall
ZeitschriftPneumologie
Ausgabe07/2015
Jahrgang / SeitenNicht angegeben
Kernaussage aus der Literatur
Abstract-Vorschau
Introduction: In CF patients, an impaired Cl-/fluid secretion across airway epithelia causing airway surface dehydration and airway mucus obstruction is due to mutations in the cystic fibrosis transmembrane conductance regulator (CFTR) gene. Previous in vivo results revealed that the epithelial chloride channel SLC26A9 is essential for preventing airway obstruction associated with IL13-induced airway inflammation. T…
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Zitierempfehlung
JJ Salomon, S Spahn, X Wang, J Füllekrug, CA Bertrand, MA Mall (2015) Alternative chloride channels in cystic fibrosis (CF): Functional characterization of SLC26A9 in stably-overexpressing FRT epithelial cells. Pneumologie (07). https://doi.org/10.1055/s-0035-1556599
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