CareLit · DOI-Nachweis · Pneumologie
Mitochondrial autophagy in the development of amiodarone induced pulmonary fibrosis.
DOI 10.1055/s-0035-1556647
Bibliografische Angaben
Nachweis in CareLit
Autor:innenS Venkatesan, S Chillappagari, I Henneke, W Seeger, S Bellusci, A Günther, P Mahavadi
ZeitschriftPneumologie
Ausgabe07/2015
Jahrgang / SeitenNicht angegeben
Kernaussage aus der Literatur
Abstract-Vorschau
Amiodarone (AD) is a potential anti-arrhythmic drug with a fatal side effect of pulmonary toxicity. Dysfunctional mitochondria and enhanced oxidative stress are well-reported observations in AD-induced pulmonary toxicity. We recently reported that AD induces autophagy in mice lungs and in mouse lung epithelial cells (MLE12) and that, such cells undergo autophagy dependent apoptosis. We now aimed to investigate if AD…
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Zitierempfehlung
S Venkatesan, S Chillappagari, I Henneke, W Seeger, S Bellusci, A Günther, P Mahavadi (2015) Mitochondrial autophagy in the development of amiodarone induced pulmonary fibrosis. Pneumologie (07). https://doi.org/10.1055/s-0035-1556647
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