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Mechanisms by which the amylin analog pramlintide reduces postprandial glucose (PPG) excursions in humans: Important role for splanchnic glucose sequestration
DOI 10.1055/s-2006-950991
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Autor:innenHJ Wörle, M Albrecht, R Linke, M Nicolaus, M Storr, J Limmer, S Zschau, C Neumann, B Göke, J Schirra
ZeitschriftZeitschrift für Gastroenterologie
Ausgabe09/2006
Jahrgang / SeitenNicht angegeben
Kernaussage aus der Literatur
Abstract-Vorschau
Pramlintide helps reduce PPG excursions by slowing gastric emptying (GE). PPG increases occur due to increases in the rate of glucose appearance. The latter is the sum of endogenous glucose production and the appearance of ingested glucose not been sequestered by the liver. Thus PPG reduction could result from greater suppression of endogenous glucose production or greater meal splanchnic sequestration. Methods: Nin…
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Zitierempfehlung
HJ Wörle, M Albrecht, R Linke, M Nicolaus, M Storr, J Limmer, S Zschau, C Neumann, B Göke, J Schirra (2006) Mechanisms by which the amylin analog pramlintide reduces postprandial glucose (PPG) excursions in humans: Important role for splanchnic glucose sequestration. Zeitschrift für Gastroenterologie (09). https://doi.org/10.1055/s-2006-950991
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