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False recognition correlates with β-Amyloid (1–42), but not with total Tau in CSF of patients with dementia or mild cognitive impairment

A Haldenwanger, H Hildebrandt · Klinische Neurophysiologie · Ausgabe 04/2008

DOI 10.1055/s-2008-1072954

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Autor:innenA Haldenwanger, H Hildebrandt
ZeitschriftKlinische Neurophysiologie
Ausgabe04/2008
Jahrgang / SeitenNicht angegeben

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Abstract-Vorschau

A severe Memory impairment forms the core symptom of Alzheimer's disease (AD) and precedes the clinical presentation of cognitive deterioration for several years. Recent studies show that Alzheimer patients not only suffer from forgetfulness, but also differ in their response bias when recognizing remembered and familiar information. Altered total Tau-protein and β-Amyloid (1–42) concentration in the CSF is also a w…

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Zitierempfehlung

A Haldenwanger, H Hildebrandt (2008) False recognition correlates with β-Amyloid (1–42), but not with total Tau in CSF of patients with dementia or mild cognitive impairment. Klinische Neurophysiologie (04). https://doi.org/10.1055/s-2008-1072954

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