CareLit Fachartikel

Studies on Mitochondrial Regulation of the Genome

Thomas Tallberg, Helene Stenbäck, Raija Hallamaa, Jan Dabek, Erland Johansson, Eeva Kallio · Deutsche Zeitschrift für Onkologie · 2002 · Heft 04 · S. 128-139

Dokument
1007205
CareLit-ID
Jahr
2002
Publikation
PDF
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Metadaten
DOI
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Bibliografische Angaben

Zeitschrift
Deutsche Zeitschrift für Onkologie
Autor:innen
Thomas Tallberg, Helene Stenbäck, Raija Hallamaa, Jan Dabek, Erland Johansson, Eeva Kallio
Ausgabe
Heft 04 / 2002
Jahrgang 34
Seiten
128-139
Erschienen: 2002-01-01 00:00:00
ISSN
1617-5891

Zusammenfassung

Summary Tumours may spontaneously regress. Consequently mammals possess a natural intrinsic regulatory capacity to control the de-differentiation of euploid cells. Bio-immunotherapy was aimed to actively simulate the biomodulating factors leading to such mysterious cures. Big tumours in mammals could re-transform into normal transcription and be cured without apoptosis or lysis following a dietary, balanced biological correction, using amino acids and essential trace element salts. The manifold long-standing aetiological metabolic deficiency, causing different cancer forms, can biologically be compensated. These…

Schlagworte

Mitochondrial function gene regulation of eukaryotic cells creation of chromosomal memory mitochondria effectors of evolution Chromosomes Apoptosis Amino Acids Computer Deutsche Zeitschrift für Onkologie