CareLit Fachartikel

Anti-LGI1 and anti-NMDA receptor encephalitis show distinct patterns of brain glucose metabolism in FDG-PET

F Wegner, E Nabavi, F Wilke, S Ben Tayeb, AL Boeck, C Trebst, M Stangel, E Voss, C Schrader, J Ahrens, A Leffler, R Rodriguez-Raecke, R Dengler, L Geworski, F Bengel, G Berding · Klinische Neurophysiologie · 2014 · Heft 03

Dokument
1113189
CareLit-ID
Jahr
2014
Publikation
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Bibliografische Angaben

Zeitschrift
Klinische Neurophysiologie
Autor:innen
F Wegner, E Nabavi, F Wilke, S Ben Tayeb, AL Boeck, C Trebst, M Stangel, E Voss, C Schrader, J Ahrens, A Leffler, R Rodriguez-Raecke, R Dengler, L Geworski, F Bengel, G Berding
Ausgabe
Heft 03 / 2014
Seiten
–
Erschienen: 2014-03-12 00:00:00
ISSN
1434-0275
DOI
10.1055/s-0034-1371187

Zusammenfassung

Background: Pathogenic autoantibodies targeting the recently identified leucin rich glioma inactivated 1 protein (LGI1) and the NR1 subunit of the N-methyl-D-aspartat (NMDA) receptor induce clinically distinct subtypes of limbic encephalitis. A comparison of brain metabolic patterns in FDG-PETs of anti-LGI1 and anti-NMDA receptor encephalitis patients has not been performed yet. Methods: The brain FDG uptake from whole-body FDG-PETs of six anti-NMDA receptor encephalitis patients and four anti-LGI1 encephalitis patients admitted to Hannover Medical School between 2008 and 2012 was retrospectively analyzed and co…

Schlagworte

Autoantibodies Klinische Neurophysiologie