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Pseudorabies virus infection stimulates pancreatic ductal HCO3- secretion

V Venglovecz, Z Rakonczay, B Ördögh, T Takács, J Lonovics, A Szabolcs, A Varró, A Tóth, G Papp, M Gray, B Argent, Z Boldogkői, P Hegyi · Zeitschrift für Gastroenterologie · 2005 · Heft 05

Dokument
1132796
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Jahr
2005
Publikation
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DOI
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Bibliografische Angaben

Zeitschrift
Zeitschrift für Gastroenterologie
Autor:innen
V Venglovecz, Z Rakonczay, B Ördögh, T Takács, J Lonovics, A Szabolcs, A Varró, A Tóth, G Papp, M Gray, B Argent, Z Boldogkői, P Hegyi
Ausgabe
Heft 05 / 2005
Seiten
–
Erschienen: 2005-05-04 00:00:00
ISSN
0044-2771

Zusammenfassung

Background. We have previously shown that hypersecretion can be observed during the early phase of experimental oedematic and necrotizing pancreatitis. However, the source and the role of hypersecretion are unknown. Several viruses have been reported to cause acute pancreatitis, but we have no information concerning the effect of viral infection on pancreatic secretion. Our aim in this study was to examine the effect of acute infection caused by a herpesvirus (pseudorabies virus; PRV) on pancreatic ductal secretion. Methods. Ba-DupGreen (BDG), a genetically modified replicating strain of PRV was used in this stu…

Schlagworte

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